Trauma

1˚ ABC
Airway
Airway is safe if pt can:
conscious and speaking in a normal tone of voice.
AW is endangered if:
if expanding hematoma or emphysema in the neck.
Intubate when:
·       GCS 8 or low
·       Breathing is gurgling / abnormal
·       Inhalation injury
If there is cervical spinal injury, handle AW 1˚, it’s more important.
·       USE ENDOTRACHEAL INRUBATION (FROM MOUTH TO TRACHEA)
·       If laryngospasm, severe maxillofacial injuries, an impacted foreign body that cannot be dislodged –> Cricothyroidomy.
·       Use a fiberoptic bronchoscope if subcutaneous emphysema in the neck → MUST: it is a sign of major traumatic disruption of the tracheobronchial tree.
Breathing
Hearing breath sounds on both sides of the chest and having satisfactory pulse oximetry establishes that breathing is okay.
Circulation
·       Insert large bore IV
·       Check if pt is in shock or not.
·       Manage Shock
Disability
·       GCS
·       Pupil
·       Neurological Sx
Exposure
Expose the pt
Rx from head to toe:
Head
Any loss of consciousness
CT
Concussion
Contusion
No focal finding
Rarely focal
No lucid interval
No lucid interval
Normal CT
Ecchymoses
No specific treatment;
observe at home for lucid interval or new focal findings
No specific treatment; observe in hospital
Subdural hematoma
Epidural hematoma
+/– focal findings
+/– focal findings
+/– lucid interval
+/– lucid interval
Venous, crescent
Arterial, biconvex or lens-shaped hematoma
Drain large ones
Drain large ones
LARGE ONES ARE?
·       Compression of ventricles or sulci
·       Herniation with abnormal breathing and unilateral dilation of the pupil
·       Worsening mental status or focal findings
If midline structures are deviated
craniotomy will help, but prognosis is bad.
 If there is no deviation
therapy is centered on preventing further damage from subsequent increased ICP.
1.     Elevate head
2.     Hyperventilate (<pco2 35)
a.     Hyperventilation is recommended when there are signs of herniation, and the goal is a PCO 2 of 35.
3.     Mannitol
How to ⤵️ O2 demand?
Sedation and hypothermia have been used to decrease brain activity and oxygen demand. Hypothermia is currently suggested as a better option to reduce oxygen demand.
Evaluation of head trauma in children age 2-18 👧:
Clinical scenario
Recommendation
Any of the following:
·       Focal neurologic findings
·       Skull fracture, especially signs of basilar skull fracture
·       Seizure
·       Persistent altered mental status (e.g., agitation, lethargy, slow response)
·       Prolonged loss of consciousness
Head CT without contrast
GCS = 15 with any of the following:
·       Vomiting
·       Headache
·       Questionable or brief loss of consciousness
·       Injury caused by high-risk mechanism of injury
·       Severe mechanism of injury
Clinical preference:
Observation for 4-6 hours
OR
Head CT without contrast
Minor head trauma (GCS = 15 with non-severe mechanism and no vomiting, headache, loss of consciousness, or signs of fracture)
No head CT

 

NECK TRAUMA

Penetrating trauma
expanding hematoma / Unstable VS / or clear signs of esophageal or tracheal injury (coughing or spitting up blood)  Surgical exploration.
Gun shot:
·      if upper areas –> arteriographic diagnosis and management
·      If base of neck –> arteriographic + enema (visualize esophagus) for diagnosis and management
Stab wounds to the upper and middle zones
Asx pts can be safely observed.
PT w/ Severe blunt trauma to the neck,
the integrity of the cervical spine has to be ascertained.
If there are neurologic deficits / or pain on palpation of cervical spine
Order CT.

Spine Trauma

General management
Prehospital:
·       Spinal immobilization (e.g., backboard, rigid cervical collar, lateral head support)
·       Careful helmet removal (e.g., motorcycle helmet)
·       Airway oxygenation
Emergency department:
·       Orotracheal intubation preferred UNLESS significant facial trauma present
·       Rapid-sequence intubation added for unconscious patients who are breathing but need ventilatory support
·       In-line cervical stabilization suggested UNLESS it interferes with intubation
·       CT of entire cervical spine
·       Monitoring for neurogenic shock from spinal cord injury
1˚ No pelvic / urethral injury
Bladder Cath
2˚ Imaging
MRI is preferred over CT.
Compression / Neurological sx
IV STEROIDS –> CT
Complete cut of SC
Nothing below lesion (no sensory nor motor)
Brown sequard (hemisection)
·      You lose 3 tracts
·      STT: contralateral loss
·       note: loss starts 2 levels below
·      DC: ipsilateral loss
·      CS: ipsilateral loss
Anterior cord
You lose motor + STT. Burst injury.
So:
·       Motor below injury is gone
·       STT: contralateral loss
Central cord
·      elderly with forced hyperextension of the neck
·      paralysis and burning pain in the upper extremities, with preservation of most functions in the lower extremities.

CHEST

 

Rib Fx
·      Rib fracture can be deadly in the elderly,
·      because of progression of pain → hypoventilation → atelectasis → pneumonia.
·      Tx: pain control.
Features
Dx
Tx
Aortic injury
·       Blunt trauma
·       Anxious pt
·       HTN / PAIN
·       CXR:
·       Mediastinal deviation
·       Mediastinal widening
CT
Anti-HTN
Surgery
Diaphragmatic
Rupture
Presents w/ either:
·       Resp distress
·       S/S of SBO
·       Abd pain referred to shoulder
CXR: bowel in chest
Surgery
Esophageal
Rupture
·       Mediastinal widening
·       Chest pain
·       crepitus & Hamman sign
·       Pleural effusion: green
Esophagogram: air leak from esophagus
·       NPO
·       IVF
·       ABx
·       Surgery 🚨
Bronchial
Rupture
·       PTX that doesn’t resolve w/ chest tube
·       Pneumomediastinum
·       Subcutaneous emphysema
CXR
Surgery
Myocardial
Rupture
Death
Myocardial
Contusion
Can presents as it pleases:
·       As MI
·       Arrhythmia
·       Wall injury
·       Temponade
·       HF
·       Most common association: sternal fx
Urgent
Echo +ECG changes
Troponin high
·       Tx based on Sx
·       If CHF: diuretics
PTX
Clear
Clear
Chest tube
PTX
Hemothorax
·      here not tension pneumothorax,
·      same finidings
·      Dx: clx –> CXR
·      Tx: tube
·      same pathophysiology, but w/ blood, and it goes down, not up.
·      Dullness on percussion
·      Dx: CXR
·      Tx:
·       Chest tube
·       YOU MUST DDx b/w lung bleeding vs. Vessel bleeding.
·       Lung bleeding stops on its own,
·       Vessels do not
·      How to ddx?
·       amount of blood, if >1500, go to OR. (thoracostomy)
Sucking Chest wound
Flail chest
جرح يدخل الهواء جوا بس مايطلعه برى.
lead to tension pneumothorax
Tx: cclusive dressing that allows air out (taped on 3 sides) but not in
كذا عظم صدر انكسر، ويصير يتحرك عكس اتجاه التنفّس، إذا سوى شهيق (تدخل)، وإذا زفير (تطلع)
issue is risk of pulmonary contusion. 
Tx: Binders  
Flail chest:
Pathophysiology
≥3 contiguous ribs fractured in ≥2 locations → flail chest segment
Findings
·       Paradoxical chest wall motion with respiration
·       Chest pain, tachypnea, rapid shallow breaths
·       CXR: rib fractures +/- contusion/hemothorax
Management
·       Pain control, supplemental oxygen
·       PPV (+/- chest tube) IF respiratory failure
Pulmonary contusion:
Clinical features
·       Present <24 hours after blunt thoracic trauma
·       Tachypnea, tachycardia, hypoxia
Diagnosis
·       Rales or decreased breath sounds
·       CT scan (most sensitive) or CXR with patchy, alveolar infiltrate NOT restricted by anatomical borders
Management
·       Pain control
·       Pulmonary hygiene (e.g., nebulizer treatment, chest PT)
·       Supplemental oxygen and ventilatory support
·       can appear up to 48 hours later
·       Dx: CXR (whitining of lungs)
·       Tx:
·       do not add fluid!
·       Diuretics.
Esophageal perforation:
?
·       Instrumentation (endoscopy)
·       Trauma
·       Boerhaave syndrome/ Esophagitis
Clx
·       Chest, back and/or epigastric pain
·       Systematic signs such as fever
·       Crepitus, Hamman sign (crunching sound auscultation)
·       Pleural effusion with atypical (e.g. green) fluid
Dx
·       CXR/CT: widened mediastinum, pneumomediastinum, PTX
·       CT: esophageal wall thickening, mediastinal fluid collection
·       Esophagography with water soluble contrast: leak from perforation
Tx
·       NPO, IV Abx and PPI
·       Emergency surgical consultation 🚨
Aortic dissection
·       ينشخل الأورطة
·       w/ deceleration injury
·       CT Angio
·       Surgery or pt die.
Abdomen
§  In patients who have blunt abdominal trauma, resuscitation must coincide with physical examination and diagnostic testing aimed at determining the presence or absence of hemoperitoneum and organ injury. Abdominal wall ecchymosis, abdominal distention, and decreased abdominal sounds may be clues to intra-abdominal injury.
§  FAST US provides an easy and rapid way to assess intra- abdominal injuries and presence of peritoneal fluid. Unstable patients should undergo a FAST examination as part of the initial assessment. Evidence of free fluid on the FAST examination is an indication to avoid diagnostic imaging and go immediately to the ORto correct the source of bleeding. CT scan is the test of choice for patients who have blunt  abdominal trauma who meet criterion for evaluation (stable).
§  Most abdominal gunshot wounds require immediate exploratory laparotomy.
§  CT scan is done in cases of blunt trauma to diagnose intra-abdominal bleeding and to identify intra-abdominal injuries if the patient is stable enough to go to the CT scanner.
§  If there is concern for internal bleeding in an unstable patient (hypotension and/or tachycardia), investigate further with an FAST scan or diagnostic peritoneal lavage.
Abdominal trauma:
GENERAL RULES:
If the patient is
            1- Peritoneal → OR (exploratory laparotomy)
            2- GIT is out → OR (exploratory laparotomy)
            3- hemodynamically unstable-FAST→ high susceptibility of internal bleeding → OR(exploratory laparotomy)
Gun shot → OR
Stab wound → follow the rule –→digital exam of the area
Blunt   follow the rule –check for hidden bleeding –FAST
                                                                                               i. → OR
                                                                                               ii.→ CT?
                                                                                                       → OR?
                                                                                                       → watch and wait (w/w)
Complications of OR
                                  1- coagulopathy – PLT + FFP
                                  2- coagulopathy + hypothermia + lactic acidosis → STOP OR 🛑
                                  3- prolonged laparotomy – Day 2 post-op –risk of compartment syndrome
Management of blunt abdominal trauma in hemodynamically unstable patients:
Focused assessment with sonography for trauma – FAST- examination
IF THE EXAMINATION
1- positive → laparotomy
2- inconclusive → DPL
                                 IF → laparotomy
                                 IF   signs of extra-abdominal hemorrhage (e.g. pelvic/ long bone fracture)?
                                                YES? → stabilize (e.g. angiography, splint)
                                                NO? → stabilize, then CT of the abdomen
3- negative → signs of extra-abdominal hemorrhage (e.g. pelvic/ long bone fracture)?
                                                YES? → stabilize (e.g. angiography, splint)
                                                NO? → stabilize, then CT of the abdomen
Pelvis
·       Pelvic hematomas are typically left alone if they are not expanding.
·       Check for other injuries (rectum, bladder, vagina, urethra “men”)
UROGENITAL
Bloody meatus
Retrograde
Compartment syndrome:
(early pain + paresthesia → S/M Sx)
Common:
·       Pain out of proportion to injury
·       Pain ⤴️  on passive stretch
·       Rapidly increasing and tense swelling
·       Paresthesia (early)
Uncommon:
·       ⤵️  sensation
·       Motor weakness (hours)
·       Paralysis (late)
·       ⤵️ distal pulses (uncommon)
Vascular
Extremity vascular trauma:
Clx
Hard signs
·       Observed bleeding
·       Presence of bruit/thrill over injury
·       Expanding hematoma
·       Signs of distal ischemia
Soft signs Δ
·       Diminished pulses
·       Bony injury
·       Neurologic 🆎
Tx
If (hard) signs or HD unstable
·       Surgical exploration
Otherwise
·       CT scan or conventional angiography
·       Duplex Doppler ultrasonography 
BURN
Burns
Features
·       Burn.
·       Associated stuff: CO / Cyanide poisoning
S/S
·       Erythema/Superficial
·       1st degree
·       Epidermis
·       + Pain
·       Blanchable
·       Superficial-partial thickness
·       2nd degree
·       Into superficial dermis
·       + Pain
·       Blanchable
·       Blisters
·       Deep-partial thickness
·       3rd degree
·       Into deep dermis
·       – Pain
·       NOT blanchable
·       Soft
·       Full thickness
·       4th degree
·       Into underlying muscle/bone
·       – Pain
·       NOT blanchable
·       Hard
Dx
·      Clx
Tx
·       ABC
·       Remove all clothing
·       if inhalational injury is suspected, best next step: immediate intubation
·       Best dx test → Bronchoscopy
·       if chemical burn, best next step: irrigation
·       if burn eschar encircles chest, best next step: escharotomy to relieve constriction
·       Fluid:
·       4*kg*BSA
·       Lenger lactate
·       1st 8 h: 1st half
·       Tetanous ppx: all patients with > 10% BSA burn or burn worse than superficial thickness need Td
·       Stress ulcer ppx: Antaacid
Comp
·       Severe burns are often complicated by wound infections and sepsis.
·       They also can present later (years later) w/ SqCC of skin.

Ophthalmology

Acute Closed-angle Glaucoma
features
Acute – emergency
S/S
·       Hx: halos around light, pain, red eye
·       On physical exam
·       the pupil is mid-dilated and does not react to light,
·       the cornea is cloudy with a greenish hue,
·       and the eye feels “hard as a rock.”
Dx
Clx – EMERGENCY
Tx
There are 2 issues:
1.     Pressure: we need to lower it down (open a hole in the eye)
2.     Dilation: we need to reverse that by ACTIVATING ALPHA & BLOCKING BETA
a.     A-agonist
b.     B-Blocker
Open Angle Glaucoma
RFs
·       Aferican-American 👨🏾‍⚖️
·       FHx
·       DM
S/S
·       initially Asx
·       Loss of PERIPHERAL VISION → progress to tunnel vision
Dx
⤴️ IOP
Ophthalmoscopy: cupping of optic n.
Tx
1˚ BB (timolol) eye drops
2˚ Laser
3˚ if still ⤴️ IOP: surgical trabeculectomy
Orbital Cellulitis
features
EMERGENCY
S/S
·      Febrile
·      Restricted eye movements
Dx
CT
Tx
I/D + IV ABx
Macular Degeneration
RF
·       ⤴️ Age
·       Smoking
S/S
·       Wet / Dry
·       Asx
·       Grid test: early signs: Straigt line appear wavy ✳️
Dx
Ophthalmoscope: drusen deposits
Tx
Dry has no tx.
Retinal Detachment
RF
·       Myopia
·       Trauma
·       Surgery
·       DM Retinopathy
(predisposing event usually occur months before Sx happens)
S/S
·       Flashes of light
·       Floaters,
·       a big dark cloud at the top of his visual field –> SEVERE
Dx
Clx
Ophthalmoscopy: Grey, elevated retina
Tx
LASER (spot welding)
DDx
Amox fogas: intermittent retinal artery occlusion
·       Presents the same as above, but “comes & goes”
Choroidal rupture
Nx
Blunt Ocular Trauma
S/S
Blurred vision following trauma
Dx
Opthalmoscopy:
·       Central scotoma
·       Edema
·       Crescent-shaped streak around optical n.
·       hemorrhage (w/ seperation of macula)
Central Retinal A. Occlusion (CROA)
features
In about 30 minutes the damage will be irreversible
S/S
·       Sudden vision loss,
·       PAINLESS
·       Cherry red spot
Dx
·       Pale optic disc
·       Cherry red fovea
Tx
🚨 1˚ OCULAR MASSAGE + O2
Chemical Burn of the eye
Dx
Hx + Clx
Tx
·       Irrigation with plain water has to be started as soon as possible wherever the injury happened; it cannot wait until arrival at the hospital.
·       Then to ER → saline irrigation & remove any particle
·       Before D/C: pH is tested to assure that no harmful chemicals remain in the conjunctival sac
Anterior Uveitis
features
·       Inflammation of uveal tract, which includes
·       iris (anterior)
·       cilliary body (anterior)
·       choroid (posterior)
·       HLA-B27
S/S
·      Redness + Pain = Anterior
·      ⤵️  Vision + floaters = Posterior
·      constricted pupil with abnormal pupil response
·      Severe photophobia
Dx
Clx
Tx
Corticosteroids
Optic neuritis
#
·       Primarily in young 👩🏻
·       Associated with MS
·       Immune-mediated demyelination
Clx
·       Acute, peaks at 2 weeks
·       Monocular vision loss
·       Eye pain with movement
·       “Washed-out” color vision
·       Afferent pupillary defect
Dx
·       MRI of the orbits & brain
Tx
·       IV corticosteroids
·       35% of cases recur
Optic Neuritis
features
·       👩🏻
·       Inflammation of optic n
·       Seen w/ MS
S/S
·       Mono-ocular LOV
·       Eye pain w/ movement
·       Washed-out” color vision
·       Afferent pupillary defect
Dx
MRI
Tx
IV Corticosteroids
Episcleritis
features
·       may be associated w/ RA
S/S
redness
Dx
phenylphrine
Tx
Self-limited

 

KERATITIS
UVEITIS
ACUTE ANGLE CLOSURE GLAUCOMA
S/S
Redness
Pain
Tearing
Photophobia
Redness
Pain
Tearing
Photophobia
Frontal headache, transient attach of blurred vision in the evening
Central vision
⤵️(distant+near)
Distant blurred
Near > disturbed
⤵️⤵️(distant + near)
Visual field
normal
floaters
Pt is too disturbed to evaluate it
Cornea
Lost corneal luster
Muddy cornea
Edematous, obscuring deeper signs
pupil
normal
Small + spastic
Fixed + dilated
AC
deep
Full of cells + flares
Shallow
IOP
normal
normal
High, rock-hard, acutely tender
Conjunctivitis
Viral
Bacterial
Allergic
#
Eye stuck in morning?
DC?
Watery
PUS, colored, thick
Watery
dischareg comes back after wiping?
Conjunctival appearance
Follicular
Nonfollicular
·       Follicular “bumpy”
·       Conjunctival edema “chemosis”
Other points
·       Viral prodrome
·       Sandy feeling
·       Burning
ø
·       Hx of allergy
·       Itching

Sebacous cell carcinoma
Ddx from Chalazion (chronic painless inflam of mebious glands) –> Bx lesion
Chalazion
·       chronic painless inflam of mebious glands
·       Tx: ABx + incision/drianage

Ped
Amblyopia
features
·       Path: Cortical blindness –> lack of visual stimulation causes the brain to just shut off the cortical input –> blindness
·       Once it’s there, it’s there
·       Multiple etiologies: Strabismus, Retinopathy of prematurity, Cong Cataracts
S/S
Depends on the etiology.
Dx
Clx
Tx
Tx the underlying  cause.
Strabismus / الحَول
features
·       Path:
·       The 2 eyes are normal, but not properly aligned the same
·       Can be devided based on etiology (easier):
o   Idiopathic
o   Refractive
o   Restrictive –> muscular (like w/ Graves)
o   Paralytic –> Neural (3rd CN palsy)
S/S
Extropia / Entropia
Dx
Clx
Tx
If at birth –> Surgery / If acquired –> Patch / Glasses.
Strabismus (ocular misalignment)
Abnormal findings
·       Constant strabismus at any age
·       Eye deviation after 4 months of age
·       Asymmetric corneal light reflexes
·       Asymmetric intensity of red reflexes
·       Deviation on cover test
·       Torticollis or head tilt
Tx options
·       Penalization therapy: Cycloplegic
·       drops to blur normal eye
·       Occlusion therapy: Patch normal eye
·       Prescription eyeglasses
·       Surgery
Complications
·       Amblyopia
·       Diplopia
Congenital Cataract
features
No red reflex
S/S
WHITE CLOUDY EYE AT BIRTH
Dx
Clx
Tx
Remove cataract
Retinoblastoma
features
·       Red reflex is not there. Instead, a white reflex
·       EMERGENCY
S/S
Leukocoria
Dx
Clx, on PEx
Tx
Surgery
Retinopathy of prematurity
features
·      Premature baby recieving high dose O2
·      Blood vessel grows abnormally on the retina
S/S
·       PEx: growths on the eye
 
Dx
Clx
Tx
·       Laser
·       F/U other complications of prematurity
·       Intraventricular hemorrhage –> US w/ Doppler
·       NEC
·       Bronchopulmonary dysplasia

ENT


For ENT Infections (OM/OE/Mastoiditis), Refer to ID from Here.

Emergencies 🚨🚑

Ludwig Angina
features
    • Abscess in the floor of the mouth,
    • Can be life-threatening
    • It involves 2 compartments: sublingual and submaxillary space bilaterally.
    • MCC is 2˚ to dental disease in the lower molars, 90% of cases
S/S
Ludwig’s angina starts as cellulitis of the submandibular region and rapidly progress to cause airway obstruction.
  • neck swelling, neck pain, odynophagia, and dysphagia
  • PEx: fever, submandibular swelling and tenderness, swelling to the floor of the mouth, tenderness to the involved teeth, stiff neck, edema in the upper part of the neck, and crepitus.
  • No LN
Complications:
  • The most life-threatening complication of Ludwig’s angina is airway obstruction.
Dx
Clx
Tx
1˚: Make sure AW is safe.
  • Early airway management is critical to the treatment of Ludwig’s angina as the most common cause of death is sudden asphyxiation from airway obstruction. Flexible fiberoptic nasal intubation is clinicians’ favored method of intubation.
  • If the patient is not able to be intubated, the next step would be an emergency tracheotomy.
2˚: Tx the Infxn
  • I/D
  • IV broad spectrum Abx

Facial n. injuries 2˚ to trauma:

    • If paralysis immediately after trauma –> result of trauma
    • If paralysis later after trauma –> swelling that will resolve spontaneously

Cavernous sinus thrombosis

    • Suspects in pt who has sinusitis and suddenly develops diplopia + fever
    • Dx: MRI
    • Tx:
      • Hospitalize
      • Tx w/ aggressive IV Abx for 3-4 weeks
      • Drain the inflammed sinus

Peds

Foreign bodies are the cause of unilateral ENT problens in toddlers. A 2-year-old with milateral
earache, tmilateral rhinorrhea, or tmilateral wheezing has a little toy truck (substitute for your favorite
toy if you wish) in his ear canal, up his nose, or into a bronchus. The appropriate endoscopy under
anesthesia will allow extraction.
Just do endoscopy w/ anesthesia & get it out.

Thyroglossal duct cysts
Midline, at hyoid bone level, moves w/ tongue. Tx: Surgery.
Brachial cleft
Along anterior border of SCM muscle
Cystic hygroma

Chronic otitis media
  • This implies that the eardrum has perforated, the perforation has failed to heal and there is ongoing infection.
  • Why it happens? 1- not adequate tx to AOM 2-Sepsis 3- general weak state
  • Complications: invasive infection – cholesteotoma – permenant deafness
  • Tx:
    • If bony → aural toilet (تنظف المكان)
  • Dry perforation → nothing
  • permenant perforation → Maryngoplasty → using temporalis fascia
🔴 COMPLICATIONS 🔴
  • can be classified into: ear / cranial complications
1️⃣Ear complications
  • Acute mastoiditis
    • FEVER + pain  (over mastoid) + pus + swelling post-auricular
    • Tx:  IMMEDIATE IV ABx → Surgery (if abscess of slow response to ABx)
  • CN7 Palsy
    • If w/ AOM → treat AOM
  • if w/ choleostotoma → Surgery
  • Cholesteotoma`
    • squamous epithelium within the middle ear. It results in accumulation of keratotic debris.
    • It expands and damages vital structures, such as dura, the facial nerve and the semicircular canals
    • Cholesteatoma is destructive and potentially lethal if untreated.
    • Tx: surgery (mastoidectomy)
Otitis media w/ effusion
  • Because some fluid in the middle ear is normal for up to several weeks after an episode of AOM, the term OME requires that the fluid be persistent for at least 3 months.
  • What are the indications or myringotomy and PE tube placement?
  1. Persistent middle ear effusion over 3 months
  2. Debilitated or immunocompromised patient
  3. More than three episodes over 6 months (especially if bilateral)
  • Complications:
    • Acute mastoiditis
      • perform CT scan of temporal bone.
      • myringotomy and IV antibiotics
    • Cholestotoma
      • tympanomastoid surgery
Epistaxis
features
    • The MC site of bleeding is an area on the nasal septum called Little’s region
    • The most common causes of epistaxis include nose picking, a foreign body in the nasal cavity, and a dry nose.
    • There’re other systematic causes.
S/S
Epistaxis
Dx
Clx
Tx
    1. Immediate measures
      • IVF if hemodynamically unstable
      • Bend forward & pinch the nostril for 5-10 m
      • Apply cold packs and sustained
      • Apply topical vasoconstrictors (e.g., oxymetazoline, phenylephrine)
    2. If epistaxis continues after 10–15 minutes
      • First-line: cauterization of the bleeding vessel using silver nitrate or electrocautery.
      • Second-line: nasal packing using gauze impregnated with paraffin and antibiotics (covering for Staphylococcus aureus).
        • Anterior epistaxis: anterior nasal packing
        • Posterior epistaxis: posterior nasal packing
    1. If epistaxis persists: arterial embolization or endoscopic ligation of the bleeding vessel
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Packing > 24 –> toxic shock syn
 types of epistaxis
Anterior epistaxis  
Posterior epistaxis  
Clx
    •  Bleeding through the anterior nasal aperture
      • Bleeding through the posterior nasal aperture
        • Bleeding is therefore not obvious but can be identified by
examining the posterior pharyngeal wall, which appears
blood-stained.
      • The patient may swallow large amounts of blood and present with
hematemesis.
Relative frequency
    •  90% of cases
    • 10% of cases
Peak incidence
    • More common among
    • children and young
    • adults
    • More common among elderly individuals
Most common site of bleeding
    • Little’s region
    • Lateral and posterior walls of the nasal cavity (Woodruffs plexus)

Choanal Atresia

    • Best initial test? Insert a catheter
    • How to confirm dx? CT Scan
    • It can be part of CHARGE
      • Colobomas (A coloboma is a hole in one of the structures of the eye, such as the iris, retina, choroid, or optic disc)
      • Heart
      • Atresia
      • Retardation
      • GU abnormalities
      • Ear abnormal
Choanal atresia
Clx
      • Unilateral (most common)
o Chronic nasal discharge
o Symptomatic during childhood
      • Bilateral
o Cyanosis that worsens with feeding & improves with crying
o Noisy breathing (stertor)
o Symptomatic shortly after birth
    • May be associated with CHARGE syndrome
Dx
Inability to pass catheter past nasopharynx
Confirmation with CT scan or nasal endoscopy
Tx
Oral airway
Surgical repair
CHARGE = Coloboma, Heart defects, Atresia choanae, growth Retardation, Genital and Ear abnormalities.

Otosclerosis
  • Bony growth of stapes that causes CONDUCTIVE hearing loss
  • Autosomal Dominant
  • Px will be in 20s w/ hearing loss & +ve FHx
  • Tx: hearing aids or surgery (romoval of stapes + putting prosthesis)
Hearing Loss
  • Before 6 m → Brainstem response
  • When 6 m → Visual reinforcement audiometry
  1. Nose:
Nasal polyps
  • What is it? pedunculated epithelial growth.
  • Why it happens? Nobody knows exactly why, but in general, they are associated with: allergic rhinitis, asthma (aspirin), CF, and chronic sinusitis.
  • Sx? obstruction sx (mouth breath, snoaring) & recurrent infections.
  • Dx? Visualization (otoscope). CT can be done.
  • Tx: Intranasal steroids → if steroid didn’t do the job, pt have multiple polyps or deformity → surgery (شيلهم وريح راسك)
Epistaxis
  • Causes: trauma (nose picking by kids), dryness (in winter),  COCAINE
  • Tx: Compress and lean forward → drugs (topical: oxymetazonolone – phenylphrine) → Anterior nasal picking
  • if bleeding site identified → Cautery.
Foreign body
  • Unilateral + purulent maladorous mucus
  • Tx: just remove it
Allergic rhinitis
  • Management:
  1. Educate pt about the condtition
  2. Avoid triggers
  3. Antihistamine
  4. Local corticosteroids  (nansonex)
  5. If severe → oral corticosteroids
  • Juvenile nasopharyngeal angiofibroma (JNA) is a benign tumor that tends to bleed and occurs in the nasopharynx of prepubertal and adolescent males. Symptoms include Nasal obstruction (80-90%), epistaxis (45-60%) – mostly unilateral and recurrent, headache (25%).
  1. Throat
  • Tonsillitis
    • Sx: Sore throat + dysphagia (children will refuse to eat), +/- drooling
    • PEx: hyperemic tonsills + cervical LN
    • Dx:
    • Tx: ABx
Tumors:
  1. Salivary gland
    • MC → Parotid (mostly benign) / Others (highly malignant)
    • Benign
      • Pleomorphic adenoma (mc tumor)
      • Warthin → associated w/ smoking + bilateral
      • Dx: FNA
      • Tx: Surgery (Gland excision w/ margin)
    • Malignant
      • Adenoid cystic carcinoma → pulmonary metastasis
    • Mucoepidermoid carcinoma (MC in children) → if epidermoid (poor prognosis) if mucoid (good prognosis)
    • Squ cell car → very aggressive (r/o metastasis to parotid LN)
    • Dx: FNA
    • Tx: 1* surgery → radiation
  2. Thyroid
    • you know it: Papillary, Follicular, Medullary and anaplastic
    • papillary prognosis in thyroid is related to size
    • U/S most sensitive
    • FNA for all (except follicular)
    • Tx: Lobectomy if small (<1.5 papillary / <4 follicular) except with FAMILIAL MEDULLARY.
  3. Oral cavity
    • High risk for malignancy
    • MCC → SCC
    • Dx: 1* line CT w/ contrast
    • Tx: SURGERY
      • +bone → chemo
  4. Laryngeal (HPV16)
    • if arise from TRUE vocal cords → glottic → hoarseness
    • if arise from FALSE vocal cords → suppra-glottic → muffled sound
    • supraglottic: otolgia, dysphagia. hemoptysis
Airway dz
  • laryngomalacia → improves when prone
  • Laryngeal papilloma → true vocal cords → hoarseness
  • conginertal subglottic stenosis
  • Croup → barking cough (parainfluenza)
    • Tx: Adrenaline inhalation + steroids
    • steeple sign on x-ray
  • Bacterial tracheitis
    • Sx: cough (barky) – stridor – sore throat – cold sx
    • PEx: Sick Sick – resp distress – FEVER
    • Dx: no response to recemic epinephrine (you thought it was croup) – GOLD STANDARD: Bronchoscopy
    • on x-ray: irregular trachea (membranes are formed)
    • Tx: 1) secure Airway (intubation may be needed) 2) Abx
  • Retropharyngeal abscess:
    • Sx of sore throat
    • neck stiffness
    • Stridor
    • on Neck x-ray ➡️ increase paravertebral space
    • Tx Intravenous broad-spectrum antibiotic / O2 or intubation
  • Grading:
    • 1: only inspiratory obstruction
    • 2: inspiratory + passive expiratory
    • 3: inspiratory + active expiratory + pulsus paradoxicus
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Notes:
  • Rhinitis medicamentosa (RM), also known as rebound rhinitis, is a condition characterized by nasal congestion that is triggered by the overuse of topical vasoconstrictive medications, most notably intranasal decongestants.
  • Quinsy Triad (Peritonsillar Abscess)
  1. Trismus (due to irritation and reflex spasm of the medial pterygoid) is the most reliable indicator of peritonsillar abscess
  2. Uvular deviation
  3. Dysphonia (“hot potato voice”)
  • lesion raised from fossa of Rosenmüller → nasopharyngeal carcinoma
  • Juvenile nasopharyngeal angiofibroma (JNA) is a benign tumor that tends to bleed and occurs in the nasopharynx of prepubertal and adolescent males.
  • In practice: Before biopsy → DO IMAGING FIRST
  • Overall, prior nasal septal surgery (septoplasty) is the most common cause of septal perforations